Studies of dietary prophylaxis in newborns at high risk for atopic disease, and provocation of skin symptoms and immunologic changes by blinded food challenges strongly suggest a significant pathogenic role for IgE-mediated FH in AD. The purpose of this research is to more precisely define the natural history and immunopathogenic mechanisms of food hypersensitivity (FH) in children with atopic dermatitis (AD).
Mechanisms of Food Hypersensitivity in Atopic Dermatitis
Objective
Abstract
Investigators
Sampson, Hugh
Institution
Mount Sinai School of Medicine
Start date
1999
End date
2001
Funding Source
Project number
5M01RR00071-360434
Categories